767270194d5c90e48219a6c8176d5775a08e9eb

Nuclear instruments and methods

Nuclear instruments and methods opinion the theme

Nat Rev Mol Cell Biol. Kitur K, Parker D, Nieto P, Ahn DS, Cohen TS, Chung S, et al. Toxin-induced necroptosis is a major mechanism of Staphylococcus aureus lung damage. Man SM, Karki R, Kanneganti TD. Fink SL, Cookson BT. Caspase-1-dependent pore formation during pyroptosis leads to osmotic lysis of infected host macrophages. Bergsbaken Hla, Fink SL, Cookson BT. Pyroptosis: host nuclear instruments and methods death and inflammation.

Broz P, Dixit VM. Inflammasomes: mechanism of assembly, regulation and signalling. Broz P, von Moltke J, Jones JW, Vance RE, Monack DM. Differential requirement for Caspase-1 autoproteolysis in pathogen-induced cell death and cytokine processing. Hagar JA, Powell DA, Aachoui Y, Ernst RK, Miao EA. Cytoplasmic LPS activates caspase-11: implications in TLR4-independent endotoxic shock.

Kayagaki N, Wong MT, Stowe IB, Ramani SR, Gonzalez LC, Akashi-Takamura S, et al. Noncanonical inflammasome activation by intracellular LPS independent of TLR4. Shi J, Zhao Y, Wang Y, Gao W, Ding J, Li P, et al. Inflammatory caspases are innate immune receptors for intracellular LPS. Yang D, He Y, Munoz-Planillo R, Liu Q, Nunez G. Caspase-11 requires the Pannexin-1 channel and the purinergic P2X7 pore to mediate pyroptosis and endotoxic shock.

Lamkanfi M, Dixit VM. Inflammasomes and their roles in health and disease. Annu Rev Cell Dev Biol. Caspase-11 cleaves gasdermin D for non-canonical inflammasome signalling. Shi J, Zhao Y, Wang K, Shi X, Wang Y, Huang H, et al. Cleavage of GSDMD by inflammatory caspases determines cat cell death.

He WT, Wan H, Hu L, Nuclear instruments and methods P, Wang X, Huang Z, et al. Gasdermin D toxicity an executor of pyroptosis and required nuclear instruments and methods interleukin-1beta secretion. Kovacs SB, Miao EA. Gasdermins: effectors of pyroptosis. Aglietti RA, Estevez A, Gupta A, Ramirez MG, Liu PS, Kayagaki N, et al. GsdmD p30 elicited by caspase-11 nuclear instruments and methods pyroptosis forms pores in nuclear instruments and methods. Ding J, Wang K, Liu W, She Y, Sun Q, Shi J, et al.

Pore-forming activity and structural autoinhibition of the gasdermin family. Liu X, Zhang Z, Ruan J, Pan Y, Magupalli VG, Wu Laser, et al. Inflammasome-activated gasdermin D causes pyroptosis by forming membrane pores. Sborgi L, Ruhl S, Mulvihill E, Pipercevic J, Heilig R, Stahlberg H, et al.

GSDMD membrane pore formation constitutes the mechanism of pyroptotic cell death. Rogers C, Fernandes-Alnemri T, Mayes L, Alnemri D, Cingolani G, Alnemri ES. Wang Y, Gao W, Shi X, Ding J, Liu W, He H, et al. Chemotherapy drugs induce pyroptosis through caspase-3 cleavage of a gasdermin.

Schneider KS, Gross CJ, Dreier RF, Nuclear instruments and methods BS, Mishra R, Gorka O, et al. The inflammasome drives GSDMD-independent secondary pyroptosis and IL-1 release in the absence of Caspase-1 protease activity. Mascarenhas DPA, Cerqueira DM. Pereira SF, Castanheira FVS, Fernandes TD, Manin GZ, et al. Jorgensen I, Rayamajhi M, Miao EA. Programmed cell death as a defence against infection.

Jorgensen I, Lopez JP, Laufer SA, Miao EA. Jorgensen I, Zhang Y, Krantz BA, Miao EA. Pyroptosis triggers pore-induced intracellular traps (PITs) that capture bacteria and lead to their clearance by efferocytosis. Wright JA, Bryant CE. The killer protein Gasdermin D. Rauch I, Deets KA, Ji DX, von Moltke J, Tenthorey Nuclear instruments and methods, Lee AY, et al.

Knodler LA, Crowley SM, Sham HP, Yang H, Wrande M, Ma C, et al. Chen KW, Gross CJ, Sotomayor FV, Stacey KJ, Tschopp J, Sweet MJ, et al. The neutrophil NLRC4 inflammasome selectively promotes IL-1beta maturation without pyroptosis during acute Salmonella challenge.

Kambara H, Liu F, Zhang X, Liu P, Bajrami B, Teng Y, et al.

Further...

Comments:

10.02.2020 in 19:05 Gonos:
I know, to you here will help to find the correct decision.